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dexamphetamine, dextroamphetamine, dextroamphetamine (adrenaline)
amphetamine (dextroamphetamine, dextroamphetamine) and methamphetamine (amphetamine,
methamphetamine is a potent CNS stimulant as used in the treatment of attention deficit hyperactivity disorder [4]. For other uses, the terms amphetamine and methamphetamine are often used interchangeably.
The stimulant effect of mescaline on dopamine release in the substantia nigra pars compacta (SNpc) of rats is similar to that produced by amphetamine, methamphetamine and dextroamphetamine.[5] Other studies have shown that mescaline and L-lysine cause no differences in dopamine responsiveness monkeys relative to amphetamine.[6][7] However, in most studies the rats are administered dextroamphetamine to produce equivalent stimulation.[8] The two amphetamines also differ in behavioral effects by affecting distinct types of synapses, which may reflect some differences in action within regions of the brain, such as prefrontal cortex (PFC). Studies indicate that mescaline may act through modulation of 5-hydroxytryptamine (serotonin) receptors PFC neurons, and thus may exert action at the brainstem level.[2]
Mescaline is a naturally occurring monoamine neurotransmitter produced by the peyote cactus (Cactaceae) in Andean
Modafinil online 100mg region of South America. It is known for its ability in the treatment of alcoholism and other addictions.
Dosage
Oral administration of 50–100 mg/kg bodyweight (up to 400 mg/kg/day) was administered for 30 consecutive days. The dosage is a dose which in humans would be expected to give 50–100 times the stimulation resulting from intravenous consumption of pure amphetamine. Mescaline is rapidly absorbed following oral absorption in the rat,[2] so that 500 mg/kg bodyweight is approximately the dose needed to produce 100 times the stimulation of intravenous (IV) use. The tolerance development process is accelerated with higher doses.
Dose should be titrated more slowly with higher doses. If a dose is not tolerated, the last dose should not exceed 3 times the initial dose, but should not last a long time. The rat's body temperature tends to drop below 34 °C after 4 h treatment has been given. It also shown in rat studies that the effects of high doses mescaline on locomotor activity, conditioned place preference and cocaine use were dose-related.[2]
The effect of M. caapi on morphine tolerance in rodents is similar to that of dextroamphetamine in humans.[9] At this dose of mescaline a single morphine was effective for 24 hours over twice as much in a non-taster and stronger narcotic. The mescaline dose is believed to be related the length of time since ingestion the plant.
Effects on mood
Mescal divinorum has demonstrated mood-altering effects when administered from 4 hours to 2 days, but these effects tended to disappear after 72 hours. Other effects of mescal divinorum are described below.[10]
Mescal divinorum used at doses of 60–120mg/kg and administered with alcohol was capable of producing effects comparable to those induced by a dose of methamphetamine or cocaine.[9]
In a study of male subjects, 3% M. potterata extract (equivalent to approximately 500 mg/kg) given orally daily for up to 14 days was able maintain mood and locomotor activity associated with abstinence for 20 days.[9]
Mescaline is capable of producing effects similar to those of cocaine and amphetamine in rats. Mescal divinorum used at doses of 60–120mg/kg administered orally for up to 14 days has been shown to maintain mood and locomotor activity associated with abstinence for up to 20 days and is capable of producing effects similar
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Psychosis
Mescal divinorum is a strong supporter of psychoactive hallucinogenic effects in humans. The acute hallucinogen effects of a gram Mexican plant mate were found to be dose‐related.[11] The subjective responses were rated as mild, vivid, and "quite powerful", with no loss of motor coordination and a "slight relaxation" associated with the experience.[11]
In a systematic review of studies on Mexican peyote (M. divinorum in the original context), an acute intoxication study was undertaken. The participants, who were given approximately 250 mg/kg of root or extract containing an estimated 80% of the maximum clinically relevant concentration (MCOC) of the active principle, were able to report subjective symptoms of intoxication after ingestion a single dose of 30 mg root extract. In the same study, a 30.
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